秦伟

Peking University Shenzhen Graduate School    Shen Zhen, China

PostdoctoralAdvisor: Professor Shuo Lin       10/2014- now

Ø Research focused on improving CRISPR/Cas9 genome editing tools in zebrafish

and constructing disease models

Ø Research focused on studying Huntington disease (HD) using zebrafish.

Peking University            Beijing, China

Doctor of Cell Biology

Advisor: Professor Shuo Lin        09/2009-07/2014

Ø Research focused on studying endoderm development in zebrafish

Dalian University of Technology               Dalian, China

Bachelor of Science in Biotechnology            09/2005-07/2009

Ø Admitted for graduate study by Peking University with the national graduate

examination waived

Ø Summer course training at Peking University

1.Wei Qin, Zelin Chen, Yihan zhang, Ruibin Yan, Guangrong yan, Song Li, Hanbing Zhong and Shuo Lin. “Nom1 Mediates Pancreas Development by Regulating Ribosome Biogenesis in Zebrafish." Plos One, 2014，9（6）：e100796-e1007964

2.Wei Qin, Fei Meng, Song Li, Hanbing Zhong and Shuo Lin. “Pgrmc1participates in zebrafish liver development by regulating liver cell proliferation through EGFR.” (2016) (manuscript in preparation)

3.Qin W, Liang F, Feng Y, Bai H, Yan R, et al. (2015) Expansion of CRISPR/Cas9 genome targeting sites in zebrafish by Csy4-based RNA processing. Cell Res 25: 1074-1077.

4.Fang Liang, Yuxiang Han, Hao Gao, Shengchang Xin, Shaodan Chen, Nan Wang, Wei Qin,Hanbing Zhong, Shuo Lin, Xinsheng Yao& Song Li. Kaempferol Identified by Zebrafish Assay and Fine Fractionations Strategy from Dysosma versipellis Inhibits Angiogenesis through VEGF and FGF Pathways. Sci. Rep. 5, 14468; doi: 10.1038/srep14468 (2015)

5.Feng Y, Chen C, Han Y, Chen Z, Lu X, Liang F, Li S,Qin W*, Lin S: Expanding CRISPR/Cas9 Genome Editing Capacity in Zebrafish Using SaCas9. G3 (Bethesda) 2016, 6(8):2517-2521.（co-corresponding author）

6.Veldman MB, Rios-Galdamez Y, Lu XH, Gu X, Qin W, Li S, Yang XW, Lin S: The N17 domain mitigates nuclear toxicity in a novel zebrafish Huntington's disease model. Mol Neurodegener 2015, 10:67